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In an interacting suggestions loop, CLOCK-BMAL1 complexes activate expression of nuclear receptors, REV-ERB ? and ROR?. Their protein products opinions to regulate BMAL1 by competitively binding retinoic acid-similar orphan receptor response aspects while in the BMAL1 promoter40. REV-ERBs repress the transcription of BMAL1, whereas RORs activate it